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Category Archives: Herbal Supplements in Kidney Disease/Failure
Hi, do you have any website about having natural diet with kidney disease, especially juice treatment or herbal?? Thanks in advance.
I am not aware of any juice diet or herbal supplements that will benefit anyone with chronic kidney disease (CKD). In the early stages of CKD, Stages 1, 2, and 3, I am a strong advocate for the DASH diet. … Continue reading →
Hello doc. Have a good day ahead. I just want to ask how can I cure my kidney failure? I got urine blood last August and late at night when I woke up to urinate until now I feel that pain on my right side. I have UTI before and spotted blood. I feel weak, can’t move and painful when I urinate. Please, need some advice what herbal medicine I will take? What to do? Food, diet? What to eat and what not to eat?
I am unable to make a specific diagnosis based on the information that you present. I recommend that you see your physician for a complete history and physical examination. I believe that you need further testing in order to establish … Continue reading →
Hi Dr. I just wanna ask if it is okay to take vitamins that boost immune system like Immunpro if you are a kidney transplant patient?
It is not a good idea to try and boost the immune system if you are taking immune suppressing drugs to keep your kidney transplant functioning. Most of these herbal supplements and vitamin preparations have not been tested with drugs … Continue reading →
My question is regarding my boyfriend, who is a type 1 diabetic and has kidney disease. In May of this year, his GFR was 80 and now today his GFR is at 18. His kidney doctor is speaking of dialysis and saying he needs to find a treatment center before his appointment in a month. He is supposed to limit potassium and phosphorus in his diet, and drink a quart and a half of fluid per day. My question is, he seems to be doing worse everyday and his face is a little swollen more and more each day. Are there any remedies that can help his kidneys function a little better such as natural remedies that he may take or drink? What do I do to help him make it until next month?
If dialysis is indicated, there are no herbal or dietary supplements that will help someone with advanced kidney disease. If he is having increasing symptoms, I suggest that your boyfriend contact his nephrologist for advice.
I am trying to go the natural route as I have stage 3 kidney disease. Is it safe for me to take boswellia? What about magnesium? And what about noni?
I have no experience with Boswellia. That herbal supplement is not familiar to me. I have no information that this compound has ever been tested in patients with chronic kidney disease (CKD). Magnesium can accumulate in patients with CKD. If … Continue reading →
Hi- History: 60 y.o female Dx’d with Crohn’s 2010. Put on Mesalamine. Developed Interstitial nephrites dx’d in 2011. Taken off Mesalamine upon which I went into Crohn’s remission for 2 years. My Creatinine which was up to 1.2 returned to .89. In 2013 Crohn’s returned. I was being treated with Infliximab for Crohn’s and then after the 13th infusion noticed an ammonia taste in my mouth. That was the only symptom. A renal panel drawn prior to the next infusion revealed a Creat. over 3 and GFR of 15, I’d like to know whether you think the following biopsy points to Crohn’s as the cause or Infliximab as the cause of this renal injury. Also, was I more likely to have interstitial nephritis again since I had it previously? Thanks. KIDNEY BIOPSY (NEEDLE) SEVERE ACUTE AND CHRONIC INTERSTITIAL NEPHRITIS, WITH AN ISOLATED NON-NECROTIZING GRANULOMA (SEE NOTE) NO EVIDENCE OF IMMUNE COMPLEX-MEDIATED OR PARAPROTEIN DEPOSITION DISEASE; CRITERIA FOR IgG4 RELATED DISEASE ARE NOT MET IN THIS SAMPLE MODERATE CHRONIC CHANGES OF THE PARENCHYMA, INCLUDING: – GLOBAL GLOMERULOSCLEROSIS (10% OF GLOMERULI) – MODERATE TUBULAR ATROPHY AND INTERSTITIAL FIBROSIS – SEVERE ARTERIAL AND ARTERIOLAR SCLEROSIS NOTE: The biopsy reveals widespread plasma cell-rich interstitial inflammation, with a focal small non-necrotizing granuloma. This type of injury is most commonly related to a hypersensitivity reaction to drugs (sulfonamides, beta-lactam and other antibiotics, anti-viral agents, diuretics, NSAIDs, cimetidine and H2-blockers, and a long list of miscellaneous drugs). Other causes of chronic active interstitial nephritis include severe various infectious processes, metabolic diseases (gout and hyperuricemic conditions), toxic processes (lithium, lead, and other heavy metals), aristolochic acid nephropathy (Chinese herb nephropathy), physical causes (obstruction and radiation injury), and other conditions (Balkan nephropathy, sarcoidosis, “idiopathic” interstitial nephritis). For most of these conditions there are no specific or even characteristic morphological findings, and the disease process can only be diagnosed by correlating the biopsy findings and the history of the use or exposure to certain drugs or substances. There is no evidence of immune complex or paraprotein deposition. IgG4 related disease is unlikely, given the results of immunohistochemistry studies (see results below). MICROSCOPIC DESCRIPTION: Sections of formalin-fixed, paraffin embedded tissue were evaluated using H&E, PAS, JMS, and trichrome stains. An H&E-stained frozen section taken from the tissue allocated for immunofluorescence microscopy and semi-thin toluidine blue-stained epoxy sections of the tissue processed for electron microscopy were also evaluated using light microscopy. The sample consists of 57 glomeruli (LM-48; IF-5; EM-4), of which 6 are globally sclerosed and several glomeruli appear hypoperfused. The non-sclerosed glomeruli are of normal size and reveal normal thickness of the glomerular capillary loops. Significant endocapillary proliferation or cellular crescents are not seen in the glomeruli. The mesangium is not significantly expanded. The interstitium reveals large areas of intense inflammation, associated with mild edema and focal tubulitis. The infiltrates are composed of lymphocytes and many plasma cells, several eosinophils and scattered neutrophils. Isolated foci of Tamm-Horsfall protein inspissation are present in the interstitium. A focal small non-necrotizing granuloma is noted, with epithelioid cells, lymphocytes, and an isolated multinucleated giant cell. In less involved areas, tubules reveal normal cellular details. In inflamed areas, tubules reveal tubulitis and focal degenerative changes. Several tubules also contain necrotic cellular debris. Several PAS-positive hyaline casts are also noted. The sample shows moderate tubular atrophy and interstitial fibrosis. Arteries and arterioles show severe sclerosis. Arterioles also show focal hyaline degeneration. IMMUNOHISTOCHEMISTRY RESULTS: The staining for CD138, IgG4, and IgG were performed using immunoperoxidase technique. Numerous CD138-positive plasma cells were identified, but only isolated cells stain for IgG4. The positive control slide and the patient’s negative non-immune control slide (normal serum) show appropriate reactivity. The immunohistochemical tests performed at Brigham and Women’s Hospital were developed and their performance characteristics determined by the Immunohistochemistry Laboratories in the Department of Pathology at BWH. They have not been cleared or approved by the U.S. Food and Drug Administration (FDA). The FDA has determined that such clearance or approval is not necessary. IMMUNOFLUORESCENCE MICROSCOPY: KIDNEY BIOPSY #: E15-926 The sections of the sample submitted for immunofluorescence studies were incubated with antibodies specific for the heavy chains of IgG, IgA, and IgM, for kappa and lambda light chains, fibrin, albumin, and complement components C3 and C1q. The sample contains 5 glomeruli. There is 1 globally sclerosed glomerulus. No significant immune deposits are seen in the glomeruli. IgM stain shows dusty reactivity in the background of the tissue; there is also fine granular reactivity for IgM (trace) along the glomerular capillary loops. Dull reactivity with fibrin is noted along the glomerular capillary loops. Tubular basement membranes show focal fine granular deposition of C3 (trace). Tubules contain several intraluminal casts reactive for polyclonal IgA. The interstitium reveals scattered fibrin deposits. Some interstitial inflammatory cells are positive for kappa or lambda light chains. The vessels exhibit focal deposition of C3. There is no difference in reactivity between kappa and lambda light chains in the glomeruli, tubular casts or background of the tissue. The immunofluorescence microscopy tests performed at Brigham and Women’s Hospital were developed and their performance characteristics determined by the Immunohistochemistry Laboratories in the Department of Pathology at BWH. They have not been cleared or approved by the U.S. Food and Drug Administration (FDA). The FDA has determined that such clearance or approval is not necessary. ELECTRON MICROSCOPY: KIDNEY BIOPSY #: E15-926 Blocks: 1 Block examined: 1 thick section; 1 thin section. The sample submitted for electron microscopy examination contains 3 glomeruli; 2 glomeruli are examined ultrastructurally. The glomerular visceral epithelial cells reveal moderate effacement of their foot processes. The glomerular basement membranes are segmentally attenuated. Morphometric analysis was performed on 105 sites, using orthogonal intercept method. The harmonic mean of the glomerular basement membrane thickness is 274 nm. The subendothelial space of the basement membrane is segmentally expanded by electron-lucent fluffy material in a few capillaries; a new layer of basement membrane material is formed under the displaced endothelium (double contours). The endothelial cells show no significant changes. The mesangium reveals normal cellular elements and a normal amount of matrix. No electron dense deposits are present in the mesangium. CLINICAL DATA: History: A 60-year-old female with Crohn’s disease who presents with AKI. BUN 44, Cr 2.79 (baseline 0.89 in 2/2014; peak 3.24 on 7/18/15), Alb 4.0, HbA1c 5.4, C3 86, C4 15, free kappa LC/lambda LC ratio 2.45, negative hepatitis B/C, negative SPEP, urine sediment WBC 4/hpf, UA blood negative, proteinuria 0.14 g/gCr. TISSUE SUBMITTED: A/1. LM. B/2. IF. C/3. EM.
I am not able to give a medical opinion without performing a complete history and physical examination. I suggest that you discuss your concerns with your nephrologist. The biopsy diagnosis appears to be an interstitial nephritis but I’m not sure … Continue reading →
I am 65 year old man and have kidney cells damaged for 14 years after going through low protein and low potassium diets. My creatinine and eGFR are OK. I have right knee cap arthritis. My question is can I take Instaflex capsule once a day.
I am not familiar with the Instaflex capsule. I looked at it online and it mostly contains herbal supplements. I would not foresee any kidney problems for this supplement, but I am unaware of any research done on patients with … Continue reading →
Hello Dr., I have CKD, my egfr is about 51-59 and my creatinine is about 1.11. I want to take magnesium, as when I do, it makes me feel good. What type and dosage should I take? Should I take it in combination with calcium and D3?
I am unable to recommend medication without performing a complete history and physical examination. I suggest that you consult with your primary care physician about taking magnesium supplements. Magnesium can be tolerated by most people but you should always check … Continue reading →
Hello Dr, My mother is suffering from kidney Disease with Diabetes, Heart problem, PD etc for many years. My mother’s creatinine is now 3.4. Please, give me a prescription to reduce creatinine at any cost. Can I use herbs like Chamomile Tea, Cinnamon Tea, Dandelion Root etc to reduce creatinine? As It is increasing day by day. I want to avoid dialysis. I want my Mother for more few days. She is the only nearest person for me. Please Help Me. May god help You. Please, give me a response.
There are no herb, teas or supplements that can be used to improve kidney function and lower the serum creatinine. Your mother should consult with a nephrologist and follow his or her advice about treatment of chronic kidney disease (CKD). … Continue reading →
I have taken astragalus for my immune system and in lieu of flu shots for almost 20 years. Today I find it on the list of supplements to avoid if one has kidney disease. My creatinine level has been 150 to 170 for several years. I have been told there was no danger in taking the Astragalus. What are your thoughts?
I have no experience with Astralgus as a food supplement in patients with chronic kidney disease (CKD). In most cases, supplements and herbal agents are on lists of substances to avoid because they have not been tested in patients with … Continue reading →